TALEN

TALEN1-apela

ID
ZDB-TALEN-140421-1
Name
TALEN1-apela
Previous Names
None
Target
Target Sequence 1
5' - TGGTCCTCATCAGCGCAG - 3'
Target Sequence 2
5' - TTGAGTAGCTTACCGTGT - 3'
Disclaimer
Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.
Note
None
Genome Resources
None
Target Location
Constructs
No data available
Genomic Features
Genomic Feature Affected Genomic Regions
a141 apela
a142 apela
vbc23 apela
Expression
Gene expression in Wild Types + TALEN1-apela
No data available
Phenotype
Phenotype resulting from TALEN1-apela
No data available
Phenotype of all Fish created by or utilizing TALEN1-apela
Phenotype Fish Conditions Figures
endoderm sox17 expression decreased amount, abnormal apelaa141/a141 standard conditions Fig. 1 with image from Norris et al., 2017
endoderm apoptotic process increased occurrence, abnormal apelaa141/a141 standard conditions Fig. 1-s1 with image from Norris et al., 2017
caudal fin mesenchyme morphology, abnormal apelaa141/a141 standard conditions Fig. 2 with image from Norris et al., 2017
ventral fin fold mesenchyme morphology, abnormal apelaa141/a141 standard conditions Fig. 2 with image from Norris et al., 2017
blastoderm fn1a expression spatial pattern, abnormal apelaa141/a141 standard conditions Fig. 7 with image from Norris et al., 2017
heart decreased width, abnormal apelaa141/a141 standard conditions Fig. 2 with image from Norris et al., 2017
blood increased accumulation caudal fin, abnormal apelaa141/a141 standard conditions Fig. 2 with image from Norris et al., 2017
endodermal cell decreased amount, abnormal apelaa141/a141 standard conditions Fig. 1 with image from Norris et al., 2017
endoderm sox17 expression decreased distribution, abnormal apelaa141/a141 standard conditions Fig. 1 with imageFig. 7 with image from Norris et al., 2017
heart decreased size, abnormal apelaa141/a141 standard conditions Fig. 2 with image from Norris et al., 2017
caudal fin blood circulation decreased occurrence, abnormal apelaa141/a141 standard conditions Fig. 2 with image from Norris et al., 2017
endoderm sox17 expression spatial pattern, abnormal apelaa141/a141 standard conditions Fig. 1 with imageFig. 7 with image from Norris et al., 2017
caudal fin morphology, abnormal apelaa141/a141 standard conditions Fig. 2 with image from Norris et al., 2017
mesodermal cell migration decreased process quality, abnormal apelaa141/a141 standard conditions Fig. 6 with imageFig. 7 with image from Norris et al., 2017
notochord posterior region malformed, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. S7 from Pauli et al., 2014
determination of liver left/right asymmetry disrupted, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. 2 from Pauli et al., 2014
heart decreased size, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. S6Fig. S7 from Pauli et al., 2014
heart agenesis, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. S6 from Pauli et al., 2014
cell migration involved in gastrulation disrupted, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. S14 from Pauli et al., 2014
blood cell increased accumulation trunk, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. 2Fig. S7 from Pauli et al., 2014
endodermal cell decreased amount, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. 2 from Pauli et al., 2014
blood circulation disrupted, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. 2 from Pauli et al., 2014
mesoderm migration involved in gastrulation disrupted, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. 2Fig. S9 from Pauli et al., 2014
hypoblast movement quality, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. S12 from Pauli et al., 2014
cell migration involved in mesendoderm migration disrupted, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. S13Fig. S15 from Pauli et al., 2014
post-vent region malformed, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. S7 from Pauli et al., 2014
extension malformed, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. S7 from Pauli et al., 2014
presumptive endoderm cell migration involved in gastrulation process quality, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. S9 from Pauli et al., 2014
whole organism dead, abnormal apelaa141/a141 (AB/TL) standard conditions text only from Pauli et al., 2014
pharyngeal endoderm malformed, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. 2 from Pauli et al., 2014
mesodermal cell accumulation margin, abnormal apelaa141/a141 (AB/TL) standard conditions Fig. S15 from Pauli et al., 2014
endoderm apoptotic process increased occurrence, abnormal apelaa142/a142 standard conditions Fig. 1-s1 with image from Norris et al., 2017
endoderm sox17 expression decreased amount, abnormal apelaa142/a142 standard conditions Fig. 1 with image from Norris et al., 2017
caudal fin mesenchyme morphology, abnormal apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
ventral fin fold mesenchyme morphology, abnormal apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
blastoderm fn1a expression spatial pattern, abnormal apelaa142/a142 standard conditions Fig. 7 with image from Norris et al., 2017
heart decreased width, abnormal apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
blood increased accumulation caudal fin, abnormal apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
endodermal cell decreased amount, abnormal apelaa142/a142 standard conditions Fig. 1 with image from Norris et al., 2017
endoderm sox17 expression decreased distribution, abnormal apelaa142/a142 standard conditions Fig. 1 with imageFig. 7 with image from Norris et al., 2017
caudal fin blood circulation decreased occurrence, abnormal apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
endoderm sox17 expression spatial pattern, abnormal apelaa142/a142 standard conditions Fig. 1 with imageFig. 7 with image from Norris et al., 2017
caudal fin morphology, abnormal apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
mesodermal cell migration decreased process quality, abnormal apelaa142/a142 control Fig. 6 with imageFig. 7 with image from Norris et al., 2017
heart decreased size, abnormal apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
mesodermal cell migration decreased process quality, abnormal apelaa141/a141; cz3331Tg standard conditions Fig. 5 with image from Norris et al., 2017
cell migration involved in gastrulation disrupted, abnormal apelaa141/a141; ha01Tg standard conditions Fig. 3 from Pauli et al., 2014
endodermal cell movement quality, abnormal apelaa141/a141; ha01Tg standard conditions Fig. S11 from Pauli et al., 2014
endoderm cell migration involved in gastrulation disrupted, abnormal apelaa141/a141; ha01Tg standard conditions Fig. S11 from Pauli et al., 2014
endodermal cell apoptotic, abnormal apelaa141/a141; ha01Tg standard conditions Fig. 1-s1 with image from Norris et al., 2017
mesodermal cell migration decreased process quality, abnormal apelaa142/a142; cz3331Tg standard conditions Fig. 5 with image from Norris et al., 2017
endoderm cell migration involved in gastrulation process quality, abnormal apelaa141/a141; cxcr4aa169/a169 standard conditions Fig. 7 with image from Norris et al., 2017
blastoderm fn1a expression spatial pattern, abnormal apelaa141/a141; cxcr4aa169/a169 standard conditions Fig. 7 with image from Norris et al., 2017
endoderm sox17 expression decreased distribution, abnormal apelaa141/a141; cxcr4aa169/a169 standard conditions Fig. 7 with image from Norris et al., 2017
endoderm sox17 expression spatial pattern, abnormal apelaa141/a141; cxcr4aa169/a169 standard conditions Fig. 7 with image from Norris et al., 2017
mesodermal cell migration decreased process quality, abnormal apelaa141/a141; cxcr4aa169/a169 standard conditions Fig. 7 with image from Norris et al., 2017
caudal fin mesenchyme morphology, abnormal apelaa141/a141; lft2a146/a146 standard conditions Fig. 2 with image from Norris et al., 2017
ventral fin fold mesenchyme morphology, abnormal apelaa141/a141; lft2a146/a146 standard conditions Fig. 2 with image from Norris et al., 2017
heart decreased width, abnormal apelaa141/a141; lft2a146/a146 standard conditions Fig. 2 with image from Norris et al., 2017
blood increased accumulation caudal fin, abnormal apelaa141/a141; lft2a146/a146 standard conditions Fig. 2 with image from Norris et al., 2017
endodermal cell increased amount, abnormal apelaa141/a141; lft2a146/a146 standard conditions Fig. 1 with image from Norris et al., 2017
endoderm sox17 expression decreased distribution, abnormal apelaa141/a141; lft2a146/a146 standard conditions Fig. 1 with image from Norris et al., 2017
caudal fin blood circulation decreased occurrence, abnormal apelaa141/a141; lft2a146/a146 standard conditions Fig. 2 with image from Norris et al., 2017
endoderm sox17 expression spatial pattern, abnormal apelaa141/a141; lft2a146/a146 standard conditions Fig. 1 with image from Norris et al., 2017
caudal fin morphology, abnormal apelaa141/a141; lft2a146/a146 standard conditions Fig. 2 with image from Norris et al., 2017
heart decreased size, abnormal apelaa141/a141; lft2a146/a146 standard conditions Fig. 2 with image from Norris et al., 2017
endoderm cell migration involved in gastrulation process quality, abnormal cxcr4aa169/a169; apelaa142/a142 standard conditions Fig. 7 with image from Norris et al., 2017
blastoderm fn1a expression spatial pattern, abnormal cxcr4aa169/a169; apelaa142/a142 standard conditions Fig. 7 with image from Norris et al., 2017
endoderm sox17 expression decreased distribution, abnormal cxcr4aa169/a169; apelaa142/a142 standard conditions Fig. 7 with image from Norris et al., 2017
endoderm sox17 expression spatial pattern, abnormal cxcr4aa169/a169; apelaa142/a142 standard conditions Fig. 7 with image from Norris et al., 2017
mesodermal cell migration decreased process quality, abnormal cxcr4aa169/a169; apelaa142/a142 standard conditions Fig. 7 with image from Norris et al., 2017
endoderm sox17 expression spatial pattern, abnormal lft2a146/a146; apelaa142/a142 standard conditions Fig. 1 with image from Norris et al., 2017
caudal fin mesenchyme morphology, abnormal lft2a146/a146; apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
ventral fin fold mesenchyme morphology, abnormal lft2a146/a146; apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
heart decreased width, abnormal lft2a146/a146; apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
blood increased accumulation caudal fin, abnormal lft2a146/a146; apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
endodermal cell increased amount, abnormal lft2a146/a146; apelaa142/a142 standard conditions Fig. 1 with image from Norris et al., 2017
endoderm sox17 expression decreased distribution, abnormal lft2a146/a146; apelaa142/a142 standard conditions Fig. 1 with image from Norris et al., 2017
caudal fin blood circulation decreased occurrence, abnormal lft2a146/a146; apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
caudal fin morphology, abnormal lft2a146/a146; apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
heart decreased size, abnormal lft2a146/a146; apelaa142/a142 standard conditions Fig. 2 with image from Norris et al., 2017
Citations